If you have a child with an ever-expanding waistline and are unsure of the cause, the answer could be hiding in the toys he plays with or the bottles she drinks from. New research published in the peer-reviewed journal, Pediatrics, suggests that both bisphenol-A (BPA), the infamously toxic plastics chemical, and phthalates could be causing young children to pack on extra pounds, which means they very well may be potential contributors to the childhood obesity epidemic currently sweeping the nation.
New research published in the peer-reviewed journal Environmental Science & Technology completely debunks the government lie that the plastics chemical bisphenol-A (BPA) is neither harmful nor persistent in humans at current exposure levels. Originating out of California, this latest assessment of fetal exposure to BPA has concluded that 100 percent of both young and unborn children now have BPA circulating in their bloodstreams, suggesting universal exposure to this ubiquitous toxin.
Working with human eggs discarded during in vitro fertilization, scientists from Boston have discovered that bisphenol A affected the egg's ability to mature and disrupted the organization and alignment of chromosomes. It is the first study to investigate effects of BPA on human egg development.
While the FDA dillydallied on reviewing its earlier ruling that bisphenol A (BPA) poses no threat to health, researchers at the National Institutes of Health — basing their findings on 2009 findings — linked BPA to the interference of brain development both in newborns and the unborn. As a result, the attorneys general of Connecticut, New Jersey and Delaware wrote a joint letter to the six primary manufacturers of baby bottles, urging them to stop using BPA in their products.
The real threat from bisphenol-A (BPA), the nefarious plastics chemical linked to endocrine disruption, may have more to do with the substances produced when the chemical is metabolized by the body rather than with the actual chemical itself. A new study published in the journal PLoS One explains how a BPA metabolite known as MPB actually tends to bind much more easily to the body's estrogen receptors than BPA does, which may help explain why some studies have overlooked BPA's tendency to wreak metabolic havoc throughout the body.